APP, the gene responsible for making beta-amyloid in the brain, may be able to reshuffle its own DNA, scientists from Sanford Burnham Prebys Medical Discovery Institute in California have claimed. This development could explain why most drugs to treat Alzheimer’s, which are designed to remove beta-amyloid protein build-ups in the brain, have proven ineffective in clinical trials.
Researchers from the University of Alabama have identified a method by which it is possible to reduce the signs of ageing, such as wrinkles and hair loss, in mice.
Scientists have long thought that regions of DNA called telomeres control how long you live. We are now learning that it is your diet and lifestyle that shape your telomeres, not the other way around.
A small population of brain cells deep in a memory-making region of the brain controls the production of new neurons and may have a role in common brain disorders.
New research presented this week at the Alzheimer’s Association International Conference (AAIC) 2018, is suggesting that the liver could be contributing to Alzheimer’s risk by failing to supply key lipids to the brain.
Researchers at Harvard Medical School have found further evidence that supports the notion that viruses could help cause the onset of Alzheimer’s disease; an idea that was once ridiculed by other sceptics and researchers.
Scientists have discovered a “big bang” of Alzheimer’s disease — the precise point at which a healthy protein becomes toxic but has not yet formed deadly tangles in the brain.
Researchers have discovered that men and women have different genetic risk factors for developing glioma.
Tiny snippets of genetic material called microRNA may offer a way to detect conditions such as Alzheimer’s disease earlier, according to a new study.
New research suggests that the level of testosterone in an adult man is largely determined by the stress they encounter during their childhood, challenging the idea that testosterone production is controlled by genetics.
Scientists seeking to unlock secrets of cellular ageing have identified a gene that triggers senescence, a phenomenon in which cells stop dividing.
Psychiatric disorders share many genetic variants, while neurological disorders appear more distinct, according to a new study from the Brainstorm Consortium.